Nutritional Therapy For Dyslipidaemia

Patients on HAART may develop abnormal serumsignificant problem for HIV-infected men and women.
lipids, including hyper-cholestero-laemia,The risk factors for developing osteopenia or
hypertriglyceridaemia and decreased HDL cholesterol,osteoporosis include genetic predisposition, being
which predisposes them to increased risk ofunderweight, having low lean body mass, not
cardiovascular disease and pancreatitis.achieving potential peak bone mass density, having an
Nutritional therapy for dyslipidaemiaaccelerated rate of bone loss, low calcium intake,
PLWHA with hyperlipidaemia should reduce theirvitamin D deficiency, inadequate or excessive protein
intake of total fat, especially satu-rated andintake, heavy alcohol use, low physical activity,
trans-fatty acids, salt and dietary cholesterol.smoking, male or female hypogonadism,
Overweight individuals should lose weight and allmalabsorption, or amenorrhea/menopause. The
PLWHA should maintain a regular aerobic exerciseaetiology of bone loss in HIV infection is unclear but
regimen. Those with hypertriglyceridaemia should alsostudies have shown increased rates of bone
limit simple carbohydrates, avoid alcohol altogether,turnover, disturbance of calcium and vitamin D
and quit smoking because it dramatically increases themetabolism, suppression of osteoblast formation, and
risk of cardiovasculardisease. Supplementation withnegative effects of high levels of cytokines. Many
omega-3 fatty acids may enhance dietary efforts toHIV-infected persons have multiple risk factors for
lower triglycerides. Although lipid parameters may notbone loss regardless of HIV infection. The
completely normalize, PLWHA may be spared thedevelopment of osteopenia or osteoporosis results in
addition of further medications to an already high pillincreased bone fragility and susceptibility to fractures
burden. It is essential to consider dietary strategieswhich further debilitates already vulnerable patients.
within the context of HIV disease, maintainingNutritional therapy for bone mineral density
adequate energy and protein intake to preventNutrition is one of the most modifiable factors in the
weight loss in susceptible individuals.development and maintenance of bone mass. People
Abnormal Glucose Tolerancewith normal bone density require 1000-1500 mg of
Abnormal glucose tolerance has been associated withelemental calcium, 400-800 IU of vitamin D, 320-420
HAART, especially protease inhibitors. Studies havemg magnesium and 1.0-1.5 g/kg protein. Those with
shown that fasting glucose may be normal but 1 hourlow bone mineral density require 1500-2000 mg
and 2 hour glucose tolerance tests are elevated. Theelemental calcium and 800-1200 IU vitamin D. Other
aetiology is not clearly defined although insulinnutrients such as vitamin K, vitamin C and zinc are
resistance appears to be the underlying mechanism.also essential for bone formation but the benefits of
PLWHA with a family history of diabetes may be atsupplementation are unproven. Smoking, alcohol,
increased risk of developing abnormal glucoseexcess salt and caffeine have also been implicated in
tolerance. The first line of therapy for hyperglycemialoss of bone mineral density. Body weight and lean
is diet and exercise.body mass should be maintained in the ideal range.
Nutritional therapy for abnormal glucose toleranceThe importance of weight-bearing exercise cannot be
PLWHA should be counselled to consume consistent,overstated. Not only does exercise stimulate bone
mixed meals (protein, carbohydrate and fat) atformation, but individuals who exercise are stronger,
regular intervals throughout the day, to limit simpleand are less susceptible to falls.Diarrhoea and
carbohydrates and increase fibre intake. Aerobicmalabsorption should be treated to enhance
exercise facilitates glycaemic control, especially afterabsorption of necessary nutrients. Osteoporosis
a meal.treatment is provided according to standard medical
Osteoporosistherapy guidelines.
Loss of bone mineral density has emerged as a