Parathyroid Glands and Bone Disease - The Interplay of Parathyroid Hormones

Like other forms of connective tissue, the bone is- Another mechanism is slower and is dependent on
composed of cells and fibers. Unlike the other typesthe activation of osteoclasts. This mechanism could
of connective tissue, however, the bone has highertake days to weeks because osteoclasts are not
amounts of calcium deposits, making it harder. Thisdirectly stimulated by parathyroid hormone. Instead,
unyielding structure of bone enables it to perform itsthey receive signals from the osteocytes to increase
functions of support and protection.bone resorption.
The physical properties of bone are nothing short of 
amazing. Aside from maintaining great strength, boneBone Disease in Hyperparathyroidism
is also capable of some degree of elasticity whileNormally, there is a strict balance maintained between
being surprisingly lightweight. From its gross structurebone deposition by osteoblasts and bone resorption
to microscopic levels, bone construction was designedby the osteoclasts. Even in mild hyperparathyroidism,
for great strength while maintaining a great economythe activity of osteoblasts can compensate for the
for material and weight.slight increase in bone resorption caused by activated
However, despite its apparent toughness, bone is aosteoclasts. However, in severe hyperparathyroidism,
living structure. It responds to metabolic andbone resorption far outweighs the formation of new
nutritional changes as well as endocrine influences.bone and the bone may be eaten away entirely. It is
Bone undergoes constant reorganization whilefor this reason that bone fractures are commonly
maintaining a certain degree of hardness necessarythe reason a hyperparathyroid patient seeks medical
for its supportive and protective functions.help.
Parathyroid HormonesIn cases of extremely high levels of parathyroid
Parathyroid hormone is responsible for controlling thehormone, radiographs of bones reveal extensive
extracellular calcium and phosphate concentrations. Itdecalcification and large punched-out areas filled with
does so by regulating intestinal reabsorption, renalosteoclasts in the form of giant cell osteoclast
excretion and the exchange between the“tumors”. This means that the bone
extracellular fluid and the bone.structure is extremely weakened by excessive
Parathyroid hormones are produced by theresorption that even slight trauma can result in
parathyroid glands located posterior to the thyroidmultiple fractures. The cystic bone disease of
gland. Normally, humans possess four parathyroidhyperparathyroidism is called osteitis fibrosa cystica.
glands but in some cases, there could be as many asPrimary hyperparathyroidism is caused by an
six. Removal of half the number of parathyroid glandsabnormality in the parathyroid glands themselves.
may not affect the normal physiology much.Usually, the increased activity of the parathyroid
However, the removal of three out of four of theglands are caused by tumors, which actively secrete
normal glands may result in transient hypothyroidism.parathyroid hormone. These tumors are more
This means that even if only one of the parathyroidcommon in women than in men.
glands remain, it can compensate for the loss of theSecondary hyperparathyroidism, on the other hand,
other three glands.occurs as a compensatory mechanism for low calcium
Parathyroid hormone acts on bone to elevate thelevels. This then signals the parathyroid glands to
levels of calcium in circulation in two ways:increase secretion of parathyroid hormone to
 replenish the depleted calcium ion in the circulation
- One is a rapid phase that calls on bone cells orwith calcium derived from deposits in the bone.
osteocytes and proceeds in a matter of minutes toSecondary hyperparathyroidism can be caused by
several hours. Existing osteocytes in the bone matrixvitamin D deficiency or chronic renal disease, in which
pump calcium ions from the bone fluid into thethe kidneys are too damaged to produce the active
extracellular fluid, increasing the circulating calcium ionsform of vitamin D in sufficient amounts.
in circulation.